M0331S,  5' Deadenylase  - 2500 units

M0331S, 5' Deadenylase - 2500 units

M0332S,  Hemo KlenTaq - 200 rxns

M0332S, Hemo KlenTaq - 200 rxns

M0332L, Hemo KlenTaq - 1000 rxns

3.705,66 RON

Hemo KlenTaq is a truncated version of Taq DNA Polymerase, lacking the first 280 amino acids. Hemo KlenTaq also contains mutations that make it resistant to inhibitors present in whole blood. It can amplify directly from whole blood from humans and mice. Hemo KlenTaq tolerates up to 20% whole blood in a 25 µl reaction (30% in 50 µl reaction).

SKU
NEB_M0332L

Hemo KlenTaq is a truncated version of Taq DNA Polymerase, lacking the first 280 amino acids. Hemo KlenTaq also contains mutations that make it resistant to inhibitors present in whole blood. It can amplify directly from whole blood from humans and mice. Hemo KlenTaq tolerates up to 20% whole blood in a 25 µl reaction (30% in 50 µl reaction).

 

Eliminate DNA extraction and purification

  • Perform PCR directly from whole blood samples
  • Bypass costly DNA extraction and purification steps
  • Amplify from limited quantities of DNA

 

Product Source

An E. coli strain that carries a mutant Taq DNA polymerase gene. The protein lacks the N-terminal 5´→ 3´ exonuclease domain and the gene has three internal point mutations.

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Price 3.114,00 RON (preturile sunt fara TVA)
Description

Hemo KlenTaq is a truncated version of Taq DNA Polymerase, lacking the first 280 amino acids. Hemo KlenTaq also contains mutations that make it resistant to inhibitors present in whole blood. It can amplify directly from whole blood from humans and mice. Hemo KlenTaq tolerates up to 20% whole blood in a 25 µl reaction (30% in 50 µl reaction).

 

Eliminate DNA extraction and purification

  • Perform PCR directly from whole blood samples
  • Bypass costly DNA extraction and purification steps
  • Amplify from limited quantities of DNA

 

Product Source

An E. coli strain that carries a mutant Taq DNA polymerase gene. The protein lacks the N-terminal 5´→ 3´ exonuclease domain and the gene has three internal point mutations.